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It was around then that the last legal challenges to calliagnosia were resolved, and we got to talking about it. We saw calli as an opportunity: what if we could live in an environment where people didn’t judge each other on their appearance? What if we could raise our children in such an environment?

The school started out being just for the children of the families in the cooperative, but other calliagnosia schools began making the news, and before long people were asking if they could enroll their kids without joining the housing co-op. Eventually we set up Saybrook as a private school separate from the co-op, and one of its requirements was that parents adopt calliagnosia for as long as their kids were enrolled. Now a calliagnosia community has sprung up here, all because of the school.

Rachel Lyons:

Tamera’s father and I gave the issue a lot of thought before we decided to enroll her there. We talked to people in the community, found we liked their approach to education, but really it was visiting the school that sold me.

Saybrook has a higher than normal number of students with facial abnormalities, like bone cancer, burns, congenital conditions. Their parents moved here to keep them from being ostracized by other kids, and it works. I remember when I first visited, I saw a class of twelve-year-olds voting for class president, and they elected this girl who had burn scars on one side of her face. She was wonderfully at ease with herself, she was popular among kids who probably would have ostracized her in any other school. And I thought, this is the kind of environment I want my daughter to grow up in.

Girls have always been told that their value is tied to their appearance; their accomplishments are always magnified if they’re pretty and diminished if they’re not. Even worse, some girls get the message that they can get through life relying on just their looks, and then they never develop their minds. I wanted to keep Tamera away from that sort of influence.

Being pretty is fundamentally a passive quality; even when you work at it, you’re working at being passive. I wanted Tamera to value herself in terms of what she coulddo , both with her mind and with her body, not in terms of how decorative she was. I didn’t want her to be passive, and I’m pleased to say that she hasn’t turned out that way.

Martin Lyons:

I don’t mind if Tamera decides as an adult to get rid of calli. This was never about taking choices away from her. But there’s more than enough stress involved in simply getting through adolescence; the peer pressure can crush you like a paper cup. Becoming preoccupied with how you look is just one more way to be crushed, and anything that can relieve that pressure is a good thing, in my opinion.

Once you’re older, you’re better equipped to deal with the issue of personal appearance. You’re more comfortable in your own skin, more confident, more secure. You’re more likely to be satisfied with how you look, whether you’re “good looking” or not. Of course not everyone reaches that level of maturity at the same age. Some people are there at sixteen, some don’t get there until they’re thirty or even older. But eighteen’s the age of legal majority, when everyone’s got the right to make their own decisions, and all you can do is trust your child and hope for the best.

Tamera Lyons:

It’d been kind of an odd day for me. Good, but odd. I just got my calli turned off this morning.

Getting it turned off was easy. The nurse stuck some sensors on me and made me put on this helmet, and she showed me a bunch of pictures of people’s faces. Then she tapped at her keyboard for a minute, and said, “I’ve switched off the calli,” just like that. I thought you might feel something when it happened, but you don’t. Then she showed me the pictures again, to make sure it worked.

When I looked at the faces again, some of them seemed different. Like they were glowing, or more vivid or something. It’s hard to describe. The nurse showed me my test results afterwards, and there were readings for how wide my pupils were dilating and how well my skin conducted electricity and stuff like that. And for the faces that seemed different, the readings went way up. She said those were the beautiful faces.

She said that I’d notice how other people’s faces look right away, but it’d take a while before I had any reaction to how I looked. Supposedly you’re too used to your face to tell.

And yeah, when I first looked in a mirror, I thought I looked totally the same. Since I got back from the doctor’s, the people I see on campus definitely look different, but I still haven’t noticed any difference in how I look. I’ve been looking at mirrors all day. For a while I was afraid that I was ugly, and any minute the ugliness was going to appear, like a rash or something. And so I’ve been staring at the mirror, just waiting, and nothing’s happened. So I figure I’m probably not really ugly, or I’d have noticed it, but that means I’m not really pretty either, because I’d have noticed that too. So I guess that means I’m absolutely plain, you know? Exactly average. I guess that’s okay.

Joseph Weingartner:

Inducing an agnosia means simulating a specific brain lesion. We do this with a programmable pharmaceutical called neurostat; you can think of it as a highly selective anesthetic, one whose activation and targeting are all under dynamic control. We activate or deactivate the neurostat by transmitting signals through a helmet the patient puts on. The helmet also provides somatic positioning information so the neurostat molecules can triangulate their location. This lets us activate only the neurostat in a specific section of brain tissue, and keep the nerve impulses there below a specified threshold.

Neurostat was originally developed for controlling seizures in epileptics and for relief of chronic pain; it lets us treat even severe cases of these conditions without the side-effects caused by drugs that affect the entire nervous system. Later on, different neurostat protocols were developed as treatments for obsessive-compulsive disorder, addictive behavior, and various other disorders. At the same time, neurostat became incredibly valuable as a research tool for studying brain physiology.

One way neurologists have traditionally studied specialization of brain function is to observe the deficits that result from various lesions. Obviously, this technique is limited because the lesions caused by injury or disease often affect multiple functional areas. By contrast, neurostat can be activated in the tiniest portion of the brain, in effect simulating a lesion so localized that it would never occur naturally. And when you deactivate the neurostat, the “lesion” disappears and brain function returns to normal.

In this way neurologists were able to induce a wide variety of agnosias. The one most relevant here is prosopagnosia, the inability to recognize people by their faces. A prosopagnosic can’t recognize friends or family members unless they say something; he can’t even identify his own face in a photograph. It’s not a cognitive or perceptual problem; prosopagnosics can identify people by their hairstyle, clothing, perfume, even the way they walk. The deficit is restricted purely to faces.

Prosopagnosia has always been the most dramatic indication that our brains have a special “circuit” devoted to the visual processing of faces; we look at faces in a different way than we look at anything else. And recognizing someone’s face is just one of the face-processing tasks we do; there are also related circuits devoted to identifying facial expressions, and even detecting changes in the direction of another person’s gaze.